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Archived from the original (PDF) on 2015-09-14

The 28-day window assumes consistent refrigeration between 3646F and protection from light exposure

Therefore, restoration of CI function bypasses defects in lipogenic and nutrient-responsive transcription networks caused by circadian disruption in adipocytes

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These findings represent an additional regulatory mechanism for ferroptosis, suggesting that prospective research could modulate ferroptosis by regulating the enzymes responsible for synthesizing membrane phospholipids containing PUFA