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In vivo and in vitro models of retinal iron overload trigger the activation of the NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome signaling pathway [21] and the synthesis and cleavage of complement C3 [22], which activates the complement system pathway, two pathogenic mechanisms implicated in AMD [11, 23]

10.1083/jcb.201708007 171 WatsonN.DingB.ZhuX.FrisinaR

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Resting-state functional connectivity revealed that the substantia nigra and all PD DBS targets (subthalamic nucleus, globus pallidus and ventral intermediate thalamus) are selectively connected to the SCAN rather than to effector-specific motor regions