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CNS Dis

And GLP-1 can inhibite hepatocyte apoptosis which induced by NLRP3 through autophagy in mitochondria, weakening the intrahepatic inflammatory response, thereby inhibiting the transition from simple fatty liver to nonalcoholic steatohepatitis (NASH)

For gut-targeted indications, it may be the superior route because high local concentrations in the GI tract are exactly what you want

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189 In addition, tumor-associated cytokines such as TNF, IL-6, and IL-1 can induce the expression of the histone demethylase JMJD1C via the NF-B and STAT3 signaling pathways, which suppress Akt and STAT3 signaling and prevents Tregs from acquiring a fragile phenotype