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A recent review of the biological rationale and evidence for considering pregabalin for the management of symptoms of CIPN concluded that pregabalin is not likely to prevent CIPN, but shows promise for treatment of established CIPN (Davis 2022)

232 Several studies have demonstrated that loss of function of UBE3A leads to increased ARC expression and subsequently decreases the number of AMPARs, ultimately impairing synaptic plasticity at excitatory synapses

In early AKI, ferroptosis-mediated elimination of irreversibly damaged PT cells may limit oxidative stress propagation and promote tissue repair, which is supported by studies showing that transient ferroptosis induction reduces AKI severity in mouse models (287, 288)

The differences between these studies may be explained by variations in the recruitment (mean age, inpatients or outpatients, studied departments) and the threshold defining the status of elevated B12

27 Hendren, R