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In preclinical models, acetyl-L-carnitine reduced brain oxidative damage markers including malondialdehyde, oxidized nucleotides, and nitrotyrosine, while L-carnitine at equivalent doses did not, despite both similarly elevating tissue carnitine levels and improving ambulatory activity.[1] Acetyl-L-carnitines superior CNS effects likely reflect its ability to donate acetyl groups for energy production, neurotransmitter synthesis, and direct antioxidant activity.[2][3] For systemic inflammation, L-carnitine demonstrates clear clinical efficacy

doi: 10.1016/j.neuron.2017.09.003 Summary Keywords apolipoproteins, fatty acid metabolism, lipid oxidation, vascular biology, Alzheimers disease Citation Huguenard CJC, Cseresznye A, Darcey T, Nkiliza A, Evans JE, Hazen SL, Mullan M, Crawford F and Abdullah L (2023) Age and APOE affect L-carnitine system metabolites in the brain in the APOE-TR model