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The first theory for the etiology of discoid meniscus was developed by Smillie in 1948 which postulated that the discoid shape is a normal stage in the developing embryo and that failure of absorption of the central portion will persist during the fetal state (congenital discoid meniscus

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Electrophysiological recordings further supported this, revealing that Dihexa caused a 1.6-fold increase and Nle-AngIV caused a 1.7-fold increase in the frequency of AMPA-mediated mEPSCs compared to vehicle-treated neurons, indicating an expansion of functional synapses without altering individual synapse properties such as amplitude, rise, or decay times

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Discontinuation Rates Retatrutide shows substantially higher discontinuation than semaglutide (18.2% vs 6.9%)