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In the FA-AKI model, FA-induced ferroptosis exacerbated renal injury by triggering inflammation to induce other types of cell death, consistent with the increase in the expression of TWEAK and Fn14 in renal tubular epithelial cells in the AKI context [164,165,166]

The liproxstatin-1 could suppress those effects induced by anti-CTLA4, further indicating the significant roles of ferroptosis in synergic treatment of radiotherapy and immunotherapy (Lang et al., 2019)