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Schneider 1 , Jan Sperl 7 , Sona Frankova 7 , Marc Bartel 8 , Holger Zimmer 9 , Markus Zorn 9 , Aleksander Krag 5 , Alice Turner 10 , Christian Trautwein 11 and Pavel Strnad 1 , (1)Medical Clinic III, Gastroenterology, Metabolic Diseases and Intensive Care, University Hospital Rwth Aachen, Aachen, Germany, (2)University Hospital Rwth Aachen, (3)Liver Unit, Vall dHebron University Hospital, Vall d`Hebron Research Institute (VHIR), Barcelona, Spain, (4)Clinic for Pneumology, Vall dHebron Hospital, Barcelona, Spain, (5)Department of Gastroenterology and Hepatology, Odense University Hospital, Odense, Denmark, (6)Department of Medicine, Gastroenterology and Hepatology Unit, Sahlgrenska University Hospital, Gothenburg, Sweden, (7)Department of Hepatogastroenterology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic, (8)Institute of Forensic and Traffic Medicine, Heidelberg University Hospital, Heidelberg, Germany, (9)Department of Internal Medicine I and Clinical Chemistry, Heidelberg University Hospital, Heidelberg, Germany, (10)University of Birmingham, Birmingham, United Kingdom, (11)University Hospital, Rwth Aachen Background: Alpha-1 antitrypsin deficiency (AATD) arises from mutations in SERPINA1 and predisposes to liver cirrhosis

doi: 10.1038/s41416-020-01156-1 55 SchiliroCFiresteinBL

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