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This data can be found at the National Center for Biotechnology Information (NCBI) using accession number PRJNA1311253

Models that express N-terminal fragments of HD exon 1 shows a rapid development of disease phenotype accompanied by motor deficits and weight loss seen in HD patients, whereas those models that contain the full-length murine Hdh gene demonstrate a lengthy disease course with attenuated motor defects and albeit specific neurodegeneration signs

204,205 Hence, zinc deficiency is crucial in the context of glutamate excitotoxicity and synaptic dysfunction in AD

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But few know that it also has profound effects on receptors in the brain