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the mechanisms exploited involve interventions on the RAS system [angiotensin-converting enzyme inhibitors (ACEIs), angiotensin II receptor blockers (ARBs)], vasodilation induced by direct vasodilators [calcium channel blockers (CCBs) such as dihydropyridine], the reduction in atrioventricular conduction (beta-blockers, CCBs such as phenylalkylamine, benzothiazepine), or diuretic actions capable of reducing electrolyte levels, particularly Na + (including loop diuretics, thiazides, and potassium sparing)

Morphological alterations and induction of oxidative stress in glial cells caused by the branched-chain alpha-keto acids accumulating in maple syrup urine disease

Alcoholic liver disease and omega-3 In vitro investigations have shown opposite effects of the main n-3 FAs on hepatocyte damage caused by ethanol, with their more significant impact on lipid rafts (49, 50)

Chromatographic separation was achieved on a 75 m inner diameter, 75 cm long EasySpray column (Thermo Fisher Scientific) using a 90-min gradient elution

J.PedersenJ.HansenC