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Their investigation revealed that upregulating GPX3 expression through the use of a GPX3 overexpression plasmid mitigated IR-induced oxidative stress

Gadaleta MN et al

Together, these findings place LDs at the center of cancer cell homeostasis and highlight their emerging potential as translational targets in cancer therapy

COMT o-methylates catechol oestrogen metabolites and mediates their detoxification, and it is plausible that the activity of this enzyme is inhibited by the accumulation of the cysteine precursor homocysteine