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In addition, autophagy is an important mechanism for regulating ferroptosis, and the m 6 A modification on the mRNAs of the autophagy-related gene BECN1 results in these m 6 A-marked mRNAs resistant to degradation after recognition by YTHDF1, contributing to dihydroartemisinin-induced hepatic stellate cell ferroptosis and conferring protection against liver fibrosis[173]

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Given the increase in the physical activity levels of the participants, their energy intake may have also increased, which could have resulted in changes in the dietary macronutrient composition