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92 Endoplasmic reticulum stress treatment mainly improves the symptoms of UC by regulating endoplasmic reticulum function and reducing the abnormal accumulation of intracellular proteins, thereby inhibiting the inflammatory response of cells

10 this form of cell death was not inhibited by caspase inhibitors but could be suppressed by treating the cells with iron-chelating agents

Carbon tetrachloride, 36,37 acetaminophen, 38 allyl alcohol, 39 ethanol, 40 and others have been suggested to involve LPO-induced cell death, with an essential role for iron