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We did not detect mutations (SNVs) in the thioredoxin reductase gene of any of the lines, indicating that auranofin resistance does not arise through modifications to TgTrxR that directly influence auranofin binding or TgTrxR enzyme activity

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Therefore, the maintenance of physiological GSH levels is crucial to protect cells from harmful molecules [1]

Plant cell Physiol

Screening and optimization of their modulators could lead to the development of medicine for modulating ferroptosis