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2022;7(69):eabi4613

doi: 10.7759/cureus.40948 42 JonesRMMercanteJWNeishAS

According to the A-tau interaction hypothesis, A and tau pathology, which results in synaptic dysfunction, neuronal death, and cognitive impairment (96), is determined by the intrinsic interaction with A and tau (A42 enhances tau phosphorylation, truncation, and aggregation, while tau can increase A synthesis)

Their high receptor specificity and relatively low toxicity profiles (compared to small molecules or full-length proteins) have made them central to 21st-century drug discovery