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In-depth studies on the mechanisms of radiation-induced fibrosis have shown that a variety of extracellular signals such as immune cells and abnormal release of cytokines, and intracellular signals such as cGAS/STING, oxidative stress response, metabolic reprogramming and proteasome pathway activation are involved in the activation of myofibroblasts

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Given the potentially broad reparative effects, IL-22 agonism represents a highly rational target in AH

The cells were resuspended in 300 L of 4:1 diluted binding buffer, and transferred to different flow tubes for another 24 h-incubation