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These findings span histological observations from the mid-20th century to recent clinical correlations and complementary animal studies, collectively supporting that eosinophils are not innocent bystanders but actively implicated in IBD pathogenesis and contributing to post-inflammatory remodeling

[52] gave diet-induced obesity (DIO) mice daily GLP-1R/Gcg receptor (GcgR) double agonist (cotadutide), Lira, Gcg agonist (g1437), and Lira+g1437 respectively

(PubMed) Eslam M, Newsome PN, Sarin SK, et al

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