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H2S biosynthesis and catabolism: new insights from molecular studies

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[107] showed that this compound promoted apoptosis by inhibiting Bcl-2 and enhancing the expressions of Bax, caspase-3, and caspase-9

This review summarized and compared the mechanisms of ferroptosis induced by JEV and HSV, and speculated that endoplasmic reticulum (ER) stress might be a prerequisite for ferroptosis in JEV and HSV-induced diseases

Tabeshpour J, Hashemzaei M, Sahebkar A