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however, it failed to prevent disease progression and demise by six and five months, respectively [41]

The glyoxalase system, FN3K, reduces the formation of precursor compounds of AGEs

Rueda, J

Animal models have shown that the increase in Th17 cells shortly preceded the increase in cytotoxic CD8 + T cells several days after TBI suggesting that Th17 cells may be responsible for cytotoxicity and neuroinflammation (159)