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It is frequently associated with high-energy gamma ( ) oscillations, which may reflect active epileptic lesions (47)

This data provides a molecular explanation for the capacity of T157P to promote disease and we hypothesize that other CMT causing mutants in the binding pocket (such as S34, S36 or K39) or within the -loop itself (P153, K161, N178, or L205) might have a similar 4HNE binding defects

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As a result, alternative approaches, such as using nutritional supplements, are being explored