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Description

Water/Aqua/Eau, Cetearyl Alcohol, Bisabolol, Zingiber Officinale (Ginger) Root Extract, Leuconostoc/Radish Root Ferment Filtrate, Glycerin, Avena Sativa (Oat) Kernel Extract, Cucumis Sativus (Cucumber) Fruit Extract, Rubus Idaeus (Raspberry) Fruit Extract, Lavandula Angustifolia (Lavender) Flower Extract, Salix Alba Bark Extract, Usnea Barbata (Lichen) Extract, Butylene Glycol , Pentylene Glycol , Hydroxyphenyl , ropamidobenzoic Acid, Citrus Medica Limonum (Lemon) Fruit Extract, Fumaria Officinalis Flower/Leaf/Stem Extract, Fumaric Acid, Echinacea Purpurea Extract, Panthenol, Acrylates/ C10-30 Alkyl Acrylate, rosspolymer, Cocamidopropyl PG-Dimonium Chloride Phosphate, Disodium Lauryl Sulfosuccinate, Cetrimonium Chloride, Aminomethyl Propanol, Glyceryl Caprylate, Disodium EDTA

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GHRP-2 Cytoprotective Evidence Base As a confirmed CD36 ligand with established PI-3K/AKT1 and PPAR pathway activity, GHRP-2 belongs to the cytoprotective GHRP subclass compounds that exhibit prosurvival, anti-inflammatory, and anti-fibrotic properties across cardiac, neuronal, and hepatic cell types via their dual GHS-R1a/CD36 pharmacology

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It was evident that decreased levels of oxidative stress in the brain were associated with treatment [74]

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This observation supports evidence from other studies, such as the work of Histopathologically, while GABA supplementation helped in preserving some aspects of hippocampal structure, significant neurodegeneration and reduced neuron density remained, particularly in the CA4 region, similar to what was observed in the PPA-treated group

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