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Notably, under the latter experimental conditions, neutrophils were found to release remarkable amounts of biologically active G-CSF, as demonstrated by the capacity of supernatants harvested from R848-treated neutrophils to trigger a G-CSF-dependent STAT3 phosphorylation in heterologous neutrophils

Single-cell and spatial multi-omics analysis reveal that targeting JAG1 in epithelial cells reduces periodontal inflammation and alveolar bone loss

This enhanced pharmacokinetic behavior was attributed to the self-assembled architecture-induced assembly induced retention (AIR) effect

and involvement in the repair of ischemia/reperfusion injuries through the inhibition of ferroptosis, as well as in the modulation of drug resistance in cancer cells

Gingival solitary chemosensory cells are immune sentinels for periodontitis