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doi: 10.3109/10826084.2015.1082595

Dysbiosis of the microbiome forms a new internal environment, under which it may affect the development of GERD from the perspectives of molecular mechanisms: microbial activation of Toll-like receptors, microbial stimulation of cyclooxygenase-2 expression, microbial stimulation of inducible nitrous oxide synthase, and activation of the NLRP3 inflammatory vesicle

Further, we analyze the methionine cycle sub-network in the Reed-Nijhout model in order to interpret experimental data in light of model predictions

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