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Loss of BCAA catabolism during carcinogenesis enhances mTORC1 activity and promotes tumor development and progression

Enriched with potent antioxidants like Astaxanthin, L-Glutathione, and Lutein, it helps protect the eyes from oxidative stress and damage caused by free radicals

Activated MAPKs phosphorylate downstream targets, resulting in the creation of inflammatory mediators and modifications in tight junction protein expression

Here, we provide a systematic discussion of these mechanism by classifying them in alterations (1) that involve steps preceding the binding of cisplatin to DNA (pre-target resistance), (2) that directly relate to DNAcisplatin adducts (on-target resistance), (3) concerning the lethal signaling pathway(s) elicited by cisplatin-mediated DNA damage (post-target resistance) and (4) affecting molecular circuitries that do not present obvious links with cisplatin-elicited signals (off-target resistance)

Jen Simmons, a former breast cancer surgeon who has transitioned into an integrative oncologist