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Besides the results from preclinical models of breast cancer shows that blocking the cancer's ability to use glutathione slowed down tumor growth Further making use of an advanced technology to search for therapies that could inhibit a tumor's ability to use glutathione, the team identified pharmacological targeting of the activity of -glutamyltransferases (GGT) using GGsTop, a putative inhibitor of GGT activity prevents the breakdown of circulating GSH leading to reduced tumour cysteine levels and slowed tumour growth In a nutshell, the findings indicate a non-canonical role for GSH in supporting tumours by acting as a reservoir of amino acids and that depriving tumours of extracellular GSH or inhibiting its breakdown could potentially prove to be a therapeutically tractable approach for patients with cancer Hecht, F., Zocchi, M., Tuttle, E.T

Mol Microbiol 53: 13071318

Exosomes derived from CAFs in PDAC do not exhibit gemcitabine resistance, however, CAFs promote chemotherapy resistance in PDAC cells by secreting exosomes and maintaining signal exchange with PDAC cells after gemcitabine treatment

Obviously I've asked to see another specialist by my doctor and they said because im now on a sustainable treatment I won't be able to see a specialist

The study also found that glutathione levels began to return to baseline approximately one month after supplementation ceased, indicating that consistent use is key to maintaining optimal levels