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FUS is known to interact with HSP60, which is associated with mitochondrial abnormalities, and these defects have been identified in FUS-FTLD patients (Deng et al., R495X dysregulates the expression of genes associated with oxidative mitochondrial metabolism in neurons, and significantly reduces the size of mitochondria, which induces neurotoxicity (Nakaya and Maragkakis, 2018)

Induction of lysosomal exocytosis and biogenesis via TRPML1 activation for the treatment of uranium-induced nephrotoxicity

He has received monthly 50g Vitamin C IVs for over seven years with stable imaging and maintained clinical well-being

Medical & affiliate disclosure This article is for educational purposes and is not medical advice

This remains an area of active research rather than established clinical practice, and until adequately powered, randomised controlled trials assess the safety and efficacy of this specific combination, clinicians and patients should approach any claims about its benefits with appropriate scepticism