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Another important factor is NF-B, whose activation represents a critical proinflammatory and proapoptotic signaling pathway involved in hyperglycemia-induced inflammation and cellular apoptosis (90)

Alarim, R

In humans, metabolic lesions (especially lipid peroxidation) and the genetic alterations underpinning them have been linked to poor hemoglobin increments in transfusion recipients 12,1618 , a measurement of transfusion efficacy that indicates increases in clinical measurements of circulating hemoglobin levels in patients requiring transfusions

doi: 10.1038/s41598-019-45282-0

Therefore, the mutation of the Y296 residue slows flavin reduction and, in terms of rate, delineates this from the binding of NADPH