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several variants of the human DRD2 have been reported to be associated with hypertension [9, 10] and disruption (D 2 / ) or renal-selective silencing of Drd2 in mice increases systolic and diastolic blood pressures [11,12,13] and may cause salt sensitivity [14]

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doi: 10.18461/ijfsd.v11i3.49

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In some embodiments, the invention encompasses a method of providing muscular therapy by contacting a cell expressing NNMT with a NNMT inhibitor (such as a cation of Formula I or IA, or otherwise disclosed herein) and one or more chemical entities (e.g., nicotinamide riboside, nicotinamide mononucleotide) that increases intracellular NAD+ levels to provide muscular therapy