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Description

The renin-angiotensin-aldosterone system excites hypothalamic paraventricular nucleus neurons in heart failure

Short-chain fatty acids modify colonic motility through nerves and polypeptide YY release in the rat

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The peptide demonstrates ability to reduce production of pro-inflammatory cytokines including tumor necrosis factor-alpha (TNF-) and interleukin-1 beta (IL-1), while promoting anti-inflammatory responses

9 Summary The available literature indicates that non-medical use of exogenous testosterone and other anabolicandrogenic steroids (AAS) is associated with a broad spectrum of multisystem adverse effects mediated by oxidative stress, mitochondrial dysfunction, endocrine disruption, and epigenetic mechanisms