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Patient subgroups, characterized by distinct symptom profiles and comorbidities, exhibited increased responses to specific treatments, e.g., a POTS-dominant cluster benefiting from autonomic modulators and a cognitive-dysfunction cluster from CNS stimulants

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hnstedt E, Tomenius HL, Vgesj E, Phillipson M

Gut dysbiosis-induced overproduction of LPS and peptidoglycans potently activates innate immune receptors, driving systemic inflammation that can breach the BBB and exacerbate neural damage (Kesika et al., 2021)

Additional information Journal peer review information: Nature Communications thanks the anonymous reviewers for their contribution to the peer review of this work