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N-acetylcysteine, which amongst many other effects, enhances the efficiency of mitochondrial energy generation has an efficacy signal in some but not all trials examining bipolar depression [60, 61]

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One plausible explanation for these results is that the gpoA mutant is unable to effectively counteract the elevated ROS concentrations encountered within the intracellular environment of host cells, despite the partial compensatory activity of the TpxD and AhpD peroxidases (Fig

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