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This interaction results in the formation of a protein complex that effectively suppresses the ubiquitination and subsequent proteasomal degradation of the transcription factor BACH1, leading to significant stabilization of BACH1 protein levels

After this, thiobarbituric acid and the homogenized mixtures of the samples were boiled at 100C for 20 min

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Phase II clinical trial demonstrating that a combination of ND-630 and a FXR agonist cilofexor reduces liver fibrosis in people with NASH