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Figure 2 2.1 Synovial fibroblasts Synovial fibroblasts play an active role in the pathogenesis of RA by undergoing various biological processes such as signal transduction, gene regulation and metabolism

The structural modifications extend plasma half-life from approximately 1020 minutes for native IGF-1 to approximately 2030 hours for IGF-1 LR3, producing a substantially different pharmacokinetic profile than the native molecule despite identical receptor pharmacology [1]

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