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Damage to axons is mediated by the following: (1) failure in mitochondrial energy metabolism due to inhibition of the respiratory chain by nitric oxide, which in turn causes a decrease in Na + /K + ATPase activity and alters Na + dependent glutamate transporters, (2) overexpression of glutamate receptors, (3) oligodendroglial excitotoxicity, (4) massive influx of extracellular Ca ++ , (5) activation of proteases, and (6) impaired axonal transport

Historically, oncology and immunology were the primary targets for pharmaceutical companies, with most resources and capital allocated to developing new technologies in these areas

The administration of m 5 C + mRNA-LNP1 yielded antibody titers similar to those elicited by canonical mRNA-LNP1

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