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doi: 10.1200/JCO.2022.40.36_suppl.384950 254 ChanATCLeeVHFHongR-LAhnM-JChongWQKimS-Bet al

1F), while the ROS and ferroptosis stresses were alleviated (Fig

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Z., & Rogero, M

These analyses revealed a marked increase in extramedullary erythropoiesis in the spleen of mice with Gpx4 -deficient hematopoiesis (Figure 2H,K), and a pronounced shift towards immature precursors cells in the BM and spleen when vitamin E was additionally depleted (Figure 2 F,L)