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For instance, in mouse embryonic stem cells, targeted disruption of glucose-6-phosphate dehydrogenase, which is the rate-limiting enzyme in the pentose phosphate pathway, the predominant metabolic pathway for the generation of NADPH (a cofactor of GR), renders cells extremely sensitive to H 2 O 2 -induced cell death 34

Conclusion: In congestive hepatopathy, mechano-stress in the liver sinusoids activates YAP/TAZ and enhances CTGF expression in LSECs, which may contribute to the development of liver fibrosis and portal hypertension

Notably, the deletion of Gclc in tumours did not affect their growth (Fig

In acute situations, the critical loss of hepatocyte compromises liver functions and eventually leads to liver failure, and liver regeneration replenishes the lost hepatocytes to maintain liver functions