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Description

Our work defines PRMT7-dependent GLUD1 methylation as a critical post-translational regulator of gastric cancer progression, though the precise mechanisms coordinating methylation and ubiquitination require further exploration

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Antitumor The TME exhibits distinctive characteristics, including hypoxia, acidic pH, and elevated levels of reduced glutathione (GSH) and H 2 O 2 , which collectively create a conducive environment for tumor proliferation, invasion, migration, adhesion, and neovascularization

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GSH-Px, Glutathione Peroxidase

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interstitial fibrosis and residual restrictive pulmonary diseases may be developed [28]

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Different studies reported that the depletion of genes encoding for the mtDNA replication machinery halts embryonic development

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