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A narrative structure therefore allowed us to organize the field around a mechanistic question - how mitochondria-centered contact failure reshapes acinar-cell injury - and to distinguish between well-supported findings, extrapolated mechanisms, unresolved controversies, and translational gaps

This result suggests that the cnd1-K658E mutation causes segregation defects by disrupting the Cnd1-Pmc4 interaction

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After being taken by mouth, traditional glutathione powder is quickly broken down by stomach acid and digestive enzymes, which means that very little of it is really absorbed