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There is a high heterogeneity of ECs surface and cytoplasmic markers [9]

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A light and electron microscopic study of experimental portal-systemic (ammonia) encephalopathy

MOTS-c has been demonstrated to disrupt the folate-methionine cycle and impede de novo purine biosynthesis, and it also regulates the expression of genes such as GLUT4, STAT3, and IL-10

During this period, there was a shift in the prevalent types of conditions