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These include neutralizing free radicals, inhibiting pro-inflammatory cytokines and mediators, improving NO bioavailability, and enhancing endothelial-dependent vasodilation [10]

Discrepancies between the two reviewers were resolved with a consultation from a third reviewer (A.B.)

Biglin ER, Price GJ, Chadwick AL, Aitkenhead AH, Williams KJ, Kirkby KJ

(2002) Curr Opin Clin Nutr Metab Care 5:321-326

Mounting evidence positions redox imbalance not merely as a secondary phenomenon but as a primary driver of the disease continuum - from initial -cell dysfunction to devastating micro- and macro-vascular complications