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However, Mac-CF BMDM had a significantly higher intracellular bacterial burden 22 hours post-PsA exposure than the Ctrl counterparts ( Supplementary Figure 2H ), indicating a bacterial killing defect in the CF macrophages in vitro

A prominent alternative viewpoint argues that the increased glucose uptake observed during neural activation is primarily metabolized via aerobic glycolysis (non-oxidative glucose consumption) (Fox et al., 1988) within the activated tissue itself, producing lactate that is largely released rather than shuttled to neurons

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