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The results showed that the expression of AMPK phosphorylated on Thr172 (pAMPK), a marker of its activation, was upregulated (Fig

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A study utilizing TCR sequencing and CITE-seq/transcriptome analysis in MM showed that greater baseline TCR diversity was associated with longer PFS, while increased clonal expansion of terminally differentiated T cell clones correlated with shorter PFS [140]

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