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These findings highlight the strong association between hypothalamic inflammation and the etiology and phenotype of PCOS, suggesting that targeting hypothalamic inflammation could be a potential therapeutic strategy (91)

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Karousis ED, Gypas F, Zavolan M, Mhlemann O

Using primary hepatocytes and peritoneal macrophages, the authors demonstrated that a mixture of 17,18-EEQ, 5-HEPE, and 9-HEPE attenuated the palmitate-induced activation of pro-inflammatory cytokines and the Jun N-terminal kinase (JNK) pathway