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Kwon HS, Koh SH

XSJ improves CDAHFD-induced liver fibrosis by inhibiting the activity of the TGF-1/Smads and PI3K/AKT/Hmox1 signaling pathways To validate the results of the transcriptomic analysis, we performed RT-qPCR to verify the selected DEGs

The redox biology of schistosome parasites and applications for drug development

The activation of AMPK broadly suppresses proinflammatory signaling pathways, including the JAK-STAT, NF-B, C/EBP, CHOP, and HIF-1 pathways, which in turn inhibits the expansion and immunosuppressive function of myeloid-derived suppressor cells (MDSCs) 42