hypothesis glutathione persister cells

hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Cysteine Depletion, a

Name: hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Cysteine Depletion, a
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Frontiers Cysteine Depletion, a Key Action to Challenge Cancer Cells to Ferroptotic Cell Death Plasticity, signaling, and metabolic rewiring in melanoma persister cells Communications Biology Glutathione peroxidase 4dependent glutathione highconsumption drives acquired platinum chemoresistance in lung cancerderived brain metastasis Liu 2021 Clinical and Translational Medicine Wiley Online Library Mechanism of Drug Tolerant Persister Cancer Cells: The Landscape and Clinical Implication for Therapy Journal of Thoracic Oncology Persister cancer cells: Iron addiction and vulnerability to ferroptosis: Molecular Cell

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have revealed that ferroptosis regulates PD-L1 expression in HNSCC through a dual mechanism: on the one hand, PD-L1 is upregulated via the ROS-driven NF-B signaling pathway (a membrane damage-independent pathway), and on the other hand, immune checkpoint molecule expression is promoted through ferroptosis stress-induced calcium influx (a membrane damage-dependent pathway)

(2009) 124:e43949

& Cambron, S

Molecules 26 (22), 6994 (2021)

633 Chronic social defeat stress-induced depressed mice showed iron accumulation and iron homeostasis imbalance in the brain, accompanied by downregulation of GSH levels and GPX4 expression, which may be a potential mechanism of lipid peroxidation and ferroptosis in depressed mice