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Here, GGT was reported to be responsible for utilization of glutathione and -glutamyl cysteine pools of host as a source of cysteine, essential in intracellular multiplication of pathogen ( ggt deletion mutant of a highly virulent F

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VEGFR-3 is the specific receptor for lymphatic growth factors VEGF-C and VEGF-D, which regulates the function of both vascular and lymphatic endothelial cells during embryonic development (36)