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Mechanistically, we determined that FA reacts with GSH, affecting the GSH:GSSG ratio and the cellular redox balance, and described a conserved mechanism to salvage GSH from FA-GSH products (HSMGSH) limiting FA cytotoxicity in human cancer cells and in C

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(106), the mTOR pathway increases GPX4 protein synthesis and sterol response element binding protein (SREBP)-mediated lipogenesis to encourage resistance to ferroptosis (107)

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MitoNEET is a protein that effectively mitigates mitochondrial iron accumulation in the presence of excess extracellular or intracellular iron [69]