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EC demonstrates relatively low and variable oral bioavailability due to rapid phase II conjugation in the small intestine and liver, coupled with extensive microbial degradation in the colon (Oketch-Rabah et al., 2020)

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nevertheless, no strong pro-surfactant C staining was evident over 12-day period, suggesting that type II pneumocytes were not present, contrasting with previous findings ( Moreover, Laboratory 2 observed that, in general, the concentration related changes induced by the benchmark chemicals ranged from increased cell degeneration, separation/detachment, multifocal thinning (e.g., Crystalline silica, Amorphous silica, and Doxorubicin) and with more severe injury locally extensive cell death/necrosis (e.g., PHMG and Akemi)