M/M polarization into pro-inflammatory (M1) and anti-inflammatory (M2) phenotypes plays a pivotal role in modulating neuronal-glial interactions, thereby influencing neuronal function, tissue repair and pain signaling
Additionally, MLIF not only influences monocytes but also participates in Th2 polarization by elevating levels of the anti-inflammatory factor IL-10 while concurrently inhibiting the synthesis of proinflammatory factors such as IL-1 and TNFs [57]
[7] Nauck, M.A., et al
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M.et al (2025)
