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Supplementary material The Supplementary material for this article can be found online at: References 1 AbeF

Indeed, HGF protects BBB disruption after microsphere embolism-induced sustained cerebral ischemia by avoiding the inhibition of Bcl-2 (Date et al., 2006), thus decreasing apoptosis, and by reducing the expression of occludin and zonula occludens (ZO)-1, which are tight junction proteins, in cerebrovascular endothelial cells (Takeo et al., 2008)

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Among these postsynaptic adhesion molecules, some have been clearly identified that bind to neurexins, such as dystroglycans, LRRTMs, calsyntenins, latrophilins and neuroligins [26, 28], which are listed in Fig